Trinkaus, V.A., Riera-Tur, I., Martínez-Sánchez, A., Bäuerlein, F.J.B., Guo, Q., Arzberger, T., Baumeister, W., Dudanova, I., Hipp, M.S., Hartl, F.U., Fernández-Busnadiego, R. (2021) In situ architecture of neuronal α-Synuclein inclusions. Nat Commun 12(1):2110. doi:


Blumenstock, S.*, Schulz-Trieglaff, E.K.*, Bolender, A.-L., Voelkl, K., Lapios, P., Lindner, J., Hipp, M.S., Hartl, F.U., Klein, R., Dudanova, I. (2020) Novel proteostasis reporter mouse reveals different effects of cytoplasmic and nuclear aggregates on protein quality control in neurons. bioRxiv doi:
*equal contribution

Blumenstock, S. and Dudanova, I. (2020) Cortical and striatal circuits in Huntington’s disease. Front Neurosci 14:82


Riera-Tur, I.*, Schaefer, T.*, Hornburg, D.*, Mishra, A., Fernández-Mosquera, L., Feigenbutz, D., Auer, P., Mann, M., Baumeister, W., Klein, R., Meissner, F., Raimundo, N., Fernández-Busnadiego, R.#, and Dudanova, I.# (2019) Amyloid-like aggregates cause lysosomal defects in neurons via gain-of-function toxicity. bioRxiv
doi: 10.1101/2019.12.16.877431
*equal contribution, #equal contribution

Burgold, J.*, Schulz-Trieglaff, E.K.*, Voelkl, K., Gutiérrez-Ángel, S., Bader, J.M., Hosp, F., Mann, M., Arzberger, T., Klein, R. #, Liebscher, S. #, and Dudanova, I. # (2019) Cortical circuit alterations precede motor impairments in Huntington’s disease mice. Sci Rep 2019 Apr 29; 9(1):6634.  doi: 10.1038/s41598-019-43024-w
*equal contribution, #equal contribution


Hosp, F. *, Gutierrez-Angel, S.*, Schaefer, M.H., Cox, J., Meissner, F., Hipp, M.S., Hartl, F.U., Klein, R., Dudanova, I.#, and Mann, M.# (2017) Spatiotemporal profiling of Huntington’s Disease inclusions reveals widespread loss of protein function. Cell Rep 21: 2291-2303
*equal contribution, #corresponding authors

Bäuerlein, F.J.B., Saha, I., Mishra, A., Kalemanov, M., Martinez-Sanchez, A., Klein, R., Dudanova, I., Hipp, M.S., Hartl, F.U., Baumeister, W., Fernandez-Busnadiego, R. (2017) In situ architecture and cellular interactions of polyQ inclusions. Cell 171: 179-187


Born, G., Grayton, H.M., Langhorst, H., Dudanova, I., Rohlmann, A., Woodward, B.W., Collier, D.A., Fernandes, C., Missler, M. (2015) Genetic targeting of NRXN2 in mice unveils role in excitatory synapse function and social behaviors. Front Synaptic Neurosci 2015 Feb 19; 7:3
doi: 10.3389/fnsyn.2015.00003

Gaitanos, T.*, Dudanova, I.*, Sakkou, M., Klein, R, Paixao, S.* (2015) The Eph Receptor Family (Book chapter) in Receptor Tyrosine Kinases: Family and Subfamilies, Edited by Deric L. Wheeler and Yosef Yarden (Springer International Publishing Switzerland), 165-264
*equal contribution, doi: 10.1007/978-3-319-11888-8_5


Schaupp, A., Sabet, O., Dudanova, I., Ponserre, M., Bastiaens, P., Klein, R. (2014) The composition of EphB2 clusters determines the strength in the cellular repulsion response. J Cell Biol 204: 409-22


Gatto, G., Dudanova, I., Suetterlin, P., Davies, A.M., Drescher, U., Bixby, J.L., Klein, R. (2013) Protein tyrosine phosphatase receptor type O inhibits trigeminal axon growth and branching by repressing TrkB and Ret signaling. J Neurosci 33: 5399-410

Dudanova, I., Klein, R. (2013) Integration of guidance cues: parallel signaling and cross-talk (Review). Trends Neurosci 36: 295-304


Dudanova, I., Kao, T.-J., Herrmann, J.E., Zheng, B., Kania, A., and Klein, R. (2012) Genetic evidence for a contribution of EphA:ephrinA reverse signaling to motor axon guidance. J Neurosci 32: 5209-5215


Dudanova, I., and Klein, R. (2011) The axon’s balancing act: cis- and trans-interactions between Ephs and ephrins. (Invited preview). Neuron 71: 1-3


Dudanova, I., Gatto, G., and Klein, R. (2010) GDNF acts as a chemoattractant to support ephrinA-induced repulsion of limb motor axons. Curr Biol 20: 2150-6


Medrihan, L., Tantalaki, E., Aramuni, G., Sargsyan, V., Dudanova, I., Missler, M., and Zhang, W. (2008) Early defects of GABAergic synapses in the brain stem of a MeCP2 mouse model of Rett syndrome. J Neurophysiol 99: 112-21


Dudanova, I., Tabuchi, K., Rohlmann, A., Südhof, T., and Missler, M. (2007) Deletion of a-neurexins does not cause a major impairment of axonal pathfinding or synapse formation. J Comp Neurol 502: 261-274


Dudanova, I.*, Sedej, S.*, Ahmad, M.*, Masius, H., Sargsyan, V., Zhang, W., Riedel, D., Angenstein, F., Schild, D., Rupnik, M., and Missler, M. (2006) Important contribution of a-neurexins to Ca2+-triggered exocytosis of secretory granules. J Neurosci 26: 10599-10613
*equal contribution

Piechotta, K., Dudanova, I., and Missler, M. (2006) The resilient synapse: insights from genetic interference of synaptic cell adhesion molecules (Review). Cell Tissue Res 326:617-42

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